http://www.wrongdiagnosis.com/s/schistosomiasis/intro.htm
Schistosomiasis: Schistosomiasis, also known as bilharzia (bill-HAR-zi-a), is a disease caused by parasitic worms. Infection with Schistosoma mansoni, S. haematobium, and S. japonicum causes illness in humans. Although schistosomiasis is not found in the United States, 200 million people are infected worldwide.

Organs Affected: bladder, intestines, liver

Types: Cutaneous Schistosomiasis, Eastern Schistosomiasis, Schistosomiasis japonica, Urinary schistosomiasis, Hepatic schistosomiasis, Schistosomiasis intercalatum, Intestinal schistosomiasis, Manson's disease, Manson's schistosomiasis, Schistosomiasis mansoni, Intestinal bilharziasis, Oriental schistosomiasis, Pulmonary schistosomiasis, Vesical schistosomiasis, Visceral schistosomiasis

List of symptoms of Schistosomiasis:
The list of symptoms mentioned in various sources for Schistosomiasis includes:

• Skin rash
• Itchy skin
• Fever
• Chills
• Cough
• Muscle aches
• Hives
• No symptoms
• Liver symptoms
  • Enlarged liver
  • Liver tenderness
• Symptoms of eggs in spinal cord:
  • Seizures
  • Paralysis
  • Spinal cord inflammation
• Gastrointestinal symptoms
  • Diarrhea
  • Blood in stool
  • Anemia
  • Dysentery
  • Hepatosplenomegaly
• Bladder infection symptoms
  • Cystitis - see also symptoms of cystitis
  • Blood in urine
  • Bladder stones

Symptoms of Schistosomiasis:
Within days after becoming infected, you may develop a rash or itchy skin. Fever, chills, cough, and muscle aches can begin within 1-2 months of infection. Most people have no symptoms at this early phase of infection.

Eggs travel to the liver or pass into the intestine or bladder. Rarely, eggs are found in the brain or spinal cord and can cause seizures, paralysis, or spinal cord inflammation. For people who are repeatedly infected for many years, the parasite can damage the liver, intestines, lungs, and bladder.

Symptoms of schistosomiasis are caused by the body's reaction to the eggs produced by worms, not by the worms themselves.

Digestive symptoms: The following list incomplete.
abdominal pain, nausea, vomiting, inability to pass gas, abdominal pain on breathing, constipation, diarrhea, abdominal sensitivity, abdominal pain near navel, abdominal pain on sneezing, abdominal pain on activity, right-side abdominal pain, abdominal pain on coughing, abdominal swelling, upper abdominal discomfort, abdominal burning, abdominal bloating, malabsorption, partial intestinal blockage, Diverticulosis

* Acute kidney failure ... nausea, vomiting, diarrhea
* Bowel Obstruction ... green vomit, constipation, fecal vomiting, vomiting, abdominal swelling

Risk Factors for Schistosomiasis
About risk factors: Risk factors for Schistosomiasis are factors that do not seem to be a direct cause of the disease, but seem to be associated in some way. Having a risk factor for Schistosomiasis makes the chances of getting a condition higher but does not always lead to Schistosomiasis. Also, the absence of any risk factors or having a protective factor does not necessarily guard you against getting Schistosomiasis.

Sources include:

• Water snails
• Drinking contaminated water
• Bathing in contaminated water
• Swimming in contaminated water
• Urine
• Feces
• Sewage
• International travel

Contagion discussion:
Infection occurs when your skin comes in contact with contaminated fresh water in which certain types of snails that carry schistosomes are living.

Fresh water becomes contaminated by Schistosoma eggs when infected people urinate or defecate in the water. The eggs hatch, and if certain types of snails are present in the water, the parasites grow and develop inside the snails. The parasite leaves the snail and enters the water where it can survive for about 48 hours.

Schistosoma parasites can penetrate the skin of persons who are wading, swimming, bathing, or washing in contaminated water. Within several weeks, worms grow inside the blood vessels of the body and produce eggs. Some of these eggs travel to the bladder or intestines and are passed into the urine or stool.

Treatments of Schistosomiasis:
Safe and effective drugs are available for the treatment of schistosomiasis. You will be given pills to take for 1-2 days.

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http://www.pokoi.com/shop/index.php?shop=1&cart=21731&cat=3&
Potassium Permanganate (KMnO4) is a dark purple crystals or granules with a metallic luster. It is the most effective way to treat swimming and farm pools. The dosage varies depending on its purpose and the chemistry of the pond water. Potassium Permanganate does not add oxygen to the water and should not be used for emergency oxygenation after dissolved oxygen depletions. However, it is also the most dangerous if not used correctly. It kills exposed pathogenic bacteria, most parasites, most fluke, fungal infections, algae, fungi (mold), and organic matter.

Potassium Permanganate also oxidizes organics in the pond, leaving the water cleaner and fresher than before treatment. One gram per one hundred gallons or one level teaspoonful per six hundred gallons. When the potassium turns amber or brown, it can be neutralized with either a water change or Hydrogen Peroxide (3% USP) one quart per five thousand gallons of pond water.

Potassium Permanganate is VERY poisonous to humans and neither it nor the water which has been treated by it should be taken internally.

3

http://www.drhuldaclark.org/ailments_Diabetes.asp
Diabetes Can be Cured

All diabetics have a common fluke parasite, Eurytrema pancreaticum, the pancreatic fluke of cattle, in their own pancreas. It seems likely that we get it from cattle, repeatedly, by eating their meat or dairy products in a raw state. It is not hard to kill with a zapper but because of its infective stages in our food supply we can immediately be reinfected.

Eurytrema will not settle and multiply in our pancreas without the presence of wood alcohol (methanol). Methanol pollution pervades our food supply -- it is found in processed food including bottled water, artificial sweetener, soda pop, baby formula and powdered drinks of all kinds including health food varieties. I presume wood alcohol is used to wash equipment used in manufacturing. If your child has diabetes, use nothing out of a can, package or bottle except regular milk, and no processed foods.

By killing this parasite and removing wood alcohol from the diet, the need for insulin can be cut in half in three weeks (or sooner!).

Be vigilant with your blood sugar checks.
The pancreas with its tiny islets that produce insulin recovers very quickly. Even if 90% of them were destroyed, requiring daily insulin shots, half of them can recover or regenerate so insulin is no longer necessary. The insulin shot itself may be polluted with wood alcohol (this is an especially cruel irony -- the treatment itself is worsening the condition). Test it yourself, using the wood alcohol in automotive fluids (windshield washer) or from a pint, as a test substance. Try different brands of insulin until you find one that is free of methanol.

Artificial sweeteners are polluted with wood alcohol! Instead of helping you cope with your diabetes, they are actually promoting it! Do not use them.

Drugs that stimulate the pancreas to make more insulin may also carry solvent pollution; test them for wood alcohol and switch brands and bottles until you find a pure one. You may not need them much longer, so the extra expense now may soon reward you.

Many persons can detoxify the amount of wood alcohol that pollutes our foods. They do not have a mold, Kojic acid, built up in their bodies as diabetics do. I have found Kojic acid in coffee, and potatoes with gray areas inside. Do not eat discolored potatoes or peels, even if cooked or baked. Being able to detoxify a poisonous substance like wood alcohol should not give us the justification for consuming it. All poisons are bad for us. Do not consume them.

All diabetic persons also carry a virus, HA virus in the pancreas. This virus is grown in the skin as a wart but is spread quite widely in the body such as in the spleen or liver besides pancreas. It is not necessary to kill the virus since it disappears when the pancreatic fluke is gone. The HA virus undoubtedly belongs to the pancreatic fluke. The question can be asked: Does the fluke or its virus cause diabetes? There might even be a bacterium, so far missed in our observations, that is the real perpetrator.

While recovering from diabetes it is very important to check your blood sugar every day. Improvement is so rapid, you may suddenly be over-insulinized by your next shot. Cut down your dose to suit your actual need.

From the book "The Cure for all Diseases" Page 173

"The Cure for all Diseases": Copyright 1995 by Hulda Regehr Clark, Ph.D., ND All rights reserved. Permission is hereby granted to make copies of any part of this document for non-commercial purposes provided the original copyright notice with the author's name is included. Published in the United States by
New Century Press,
2232 Verus Street, Suite D, San Diego, CA. 92154,
(619) 423-7500, (800) 519-2465

4

http://www.path.cam.ac.uk/~schisto/OtherFlukes/Human.flukes.html
Helminthology and General Parasitology Pages, Cambridge Univ., UK
Digenean Flukes Causing Human Disease

... A number of flukes that normally infect animals may occasionally infect man accidentally ... some flukes may give rise to transient, pseudoinfections, usually of the throat.

Fasciolopsis buski (Giant Intestinal Fluke)
... The lifecycle is very similar to that of F. hepatica. The unembryonated eggs are passed in the faeces, and if reaching water begin to embryonate (takes ~3 - 7 weeks at 80 - 90° F). The egg hatches when fully developed, releasing the miracidium, which then find and penetrate suitable snail intermediate hosts (e.g. Segmentina sp.). Asexual development takes place in the snail with generations of sporocysts, redia then finally cercaria.

Cercaria are released from the snail, which then encyst as metacercaria on water vegetation, particularly Trapa natans (the water caltrop) or the water chestnut, which thrives in ponds fertilised by "night soil" (human faeces). Only the inner part of these plants are eaten, but infection of man occurs if the outer covering of the infected plant is peeled using the persons teeth. Metacercaria excyst in the duodenum, maturing to adults in ~ 3 months.

The first symptoms appear towards the end or the incubation periods, when the flukes reach maturity. Initially there may be inflammation and later ulceration at the site of attachment, eventually leading to haemorrhage and/or ulceration. Other symptoms may include a toxic diarrhoea with hunger pains, generalised toxic and allergic symptoms such as oedema of the face and abdominal wall, and generalised abdominal pain, nausea and vomiting. The skin may also eventually become very dry and harsh. Death may occur due to profound intoxication due to absorption by the host of fluke metabolites.

Metagonimus yokogawi
... this parasite also infects a wide variety of other mammals as well, including most carnivores, pigs, mice and birds such as the pelican.

Lifecycle
The eggs are almost certainly ingested by the snail intermediate hosts (of the genera Semisulcospira, Thiara and Hua, the miracidium hatching in the snails intestine. There then follows a number of asexual stages consisting of sporocyst, two generations of redia, and finally cercaria. The cercaria, which have elongate spinous bodies and long tails, emerge from the snails, then penetrate the freshwater fish second intermediate hosts, (usually Salmonids or Cyprinoids), encysting as metacercaria in the scales, tail, fins or gills.

The definitive host [including humans] is infected by ingestion of raw fish, the adult parasites being found embedded in the wall of the jejunum, ileum, caecum and, rarely, the duodenum.

Quite heavy infections often occur, with thousands of parasites, where there may be digestive disturbances and diarrhoea. More normally the infections are either asymptomatic, or the symptoms are negligible.

Gastrodiscoides hominis
... The adult flukes are located in the jejunum and upper ileum in man, as well as a number of animal zoonotic hosts, including cats, dogs, foxes and other fish eating mammals.

Lifecycle
Eggs are passed in the faeces, but to continue the lifecycle must be ingested by suitable snail intermediate hosts (e.g. Pirenella conica in Egypt, or Cerithidia cigulata in Japan).. Once ingested the eggs hatch releasing the miracidium, which then develops to the sporocyst stage, followed by primary (and sometimes secondary) generations of redia, then finally the cercarial stage (which morphologically are lophocercous - i.e. with a "keeled" tail). Cercaria seek out and penetrate a second intermediate fish host (e.g. Tilapia), where they encyst as metacercaria. The definitive host (e.g. man) is infected by eating raw infected fish.

[Symptoms]
... Mild inflammatory reactions may occur where they attach to the intestinal wall, giving rise to colic, excessive mucous production and diarrhoea. More severe pathology may occasionally be due to the eggs, which may pass to the mesenteric lymphatics or heart musculature, where tissue reactions may occur.

[Cautions]
In addition to the human parasites detailed in these pages a number of other species normally infecting other animals have been reported in man. In addition the cercaria of a number of related parasites may penetrate the skin of man, where they die, causing cercarial dermatitis.

[Accidental Infections]
Dicrocoelium dendriticum: Man may be infected by accidental infection of ants which act as second intermediate hosts, containing infective metacercaria.

Apophallus donicum: The adult parasite is usually found in small intestine of fish eating mammals, metacercaria being found in the fresh water fish second intermediate host. ... it is found in Europe and North America.

Metorchis albidus: The adult parasite is usually found in fish-eating mammals such as cats and dogs as the second intermediate hosts are fish such as the silver bream. It is found in Northern temperate and Arctic regions.

... Last Modified on 5th October 1998

5

http://www.path.cam.ac.uk/~schisto/OtherFlukes/Paragonimus.html
Helminthology and General Parasitology Pages, Cambridge Univ., UK
Paragonimus westermani (The Lung Fluke)

Digenean eggs are operculate, and contain a miracidium larvae. This egg, of the lung fluke, is morphologically a typical example of a Digenean egg. However, it should be noted that in this particular parasite, the eggs are found in the sputum as well as the faeces (as a consequence of the location of the adult flukes in the lungs), those in the faeces having been swallowed. Most Digenean eggs are found only in the faeces. Paragonimus westermani is the most well known species infecting man (in addition to zoonotic infections in cats, dogs, pigs and a large number of other animals). However the genus includes a large number of species, all of which also appear to be capable of infecting man as well.

Lifecycle
The adult parasites normally live in the lungs of the definitive host, though ectopic parasites found in other locations are also frequently seen, suggesting this parasite is not well adapted to living in man. Unembryonated eggs are laid by the parasites, and are passed either directly in the sputum, or may be swallowed then passed in the faeces. The miracidium develops over 17 - 21 days at 30°C, then hatches in water to infect the snail intermediate host by penetration.

These snail hosts belong to a number of genera depending on the geographical location of the parasite, but the principle host are species of the genus Semisulcuspira (e.g. S. libertina). The parasite then undergoes a series of asexual divisions from sporocyst, to two generations of redia, then finally the cercaria. Morphologically these are unusual having only a small (microcercus) tail, a spiny tegument and a stylet.

These cercaria, which can survive up to 48 hours, do not swim to find their next host, but either crawl in a leech like manner or are carried by currents in the water. The second intermediate hosts for this parasite are fresh-water crustaceans, particularly crabs. Once the cercaria locates these it penetrates them, and migrates to the muscles or heart where they encyst as metacercaria, where they are infective after ~ 6 weeks.